What is Pulmonary Oedema –
Pulmonary edema is fluid accumulation in the tissue and air spaces of
the lungs leading to impaired gas exchange which may cause respiratory failure.
It is due to either failure of the left ventricle of the heart to
remove blood adequately from the pulmonary circulation (cardiogenic pulmonary
edema), or an injury to the lung parenchyma or vasculature of the lung
(noncardiogenic pulmonary edema).
Discussion
The most common symptom of pulmonary edema is presents with difficulty
in breathing, but may include other symptoms such as coughing up blood ( seen
as pink, frothy sputum), excessive sweating, anxiety, and pale skin, orthopnea
(inability to lie down flat due to breathlessness) and/or paroxysmal nocturnal
dyspnea (episodes of severe sudden breathlessness at night).
These are common presenting symptoms of chronic pulmonary edema due to
left ventricular failure. The development of pulmonary edema may be associated
with symptoms and signs of "fluid overload"; this is a non-specific
term to describe the manifestations of left ventricular failure on the rest of
the body and includes peripheral edema (swelling of the legs, in general, of
the "pitting" variety, wherein the skin is slow to return to normal
when pressed upon), raised jugular venous pressure and hepatomegaly, where the
liver is enlarged and may be tender or even pulsatile. Other signs include
end-inspiratory crackles (sounds heard at the end of a deep breath) on
auscultation and the presence of a third heart sound.
As, the prakruti shift towards parthiv
which is else parthivo jalaja. The tissue becomes hygroscopic and also finds a
lot of akash element providing space for jal accumulation as fibrosis ensues. The
lung tissue which retains water possibly due to impaired drainage across the
capillary channels and hygroscopic character which consequently results in
oedema. The important point to be noted is –
Loss of heat symbolic of reduced pitta
and also shows up in reduced drainage as along with it pulmonary dosha balance
is symbolic of a dried state which is shown up as fibrosed tissue. We must
remember that as the heart is cycles the blood with each pulse, the same cycles
across the pulmonary channels. This shift reduces the pulmonary partial
pressure and affects exchanges and also increases cardiac load which in turn
leads to pulmonary oedema in a cyclic change.
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